ch 6: MI -exercise management for persons with chronic diseases/disabilities

Coronary artery disease (CAD)
the vessels that supply the heart with blood become progressively narrower
myocardial ischemia
partial blockage by plaque ( may result to MI)
myocardial infarction
complete blockage (heart attack)
occluded
blocked
other factors that help determine the severity of CAD
hemorrhage/rupture
calcification- blood vessel hardens
thrombrosis- clotting
type of plaque
reducing the risks of AMI (acute) and MI
lower LDL
reduce certain inflammatory markers (CRP- reactive protein)
increase HDL
S/S of MI
severe, prolonged pain (chest, neck, arm, back, jaw)
sweating, nausea, vomiting
increased cardiac enzyme concentration
alterations in EKG activity
left ventricular necrosis
decreases contractility
SV impaired
HR increased
intrinsic diseases of SA and AV nodes
decreases HR
decreases SV
cardiovascular meds for patients who have had MI
diuretics-slow HR
beta blockers, alpha blockers
vasodilators
angiotensin-converting enzyme (ACE) inhibitors
angiotensin receptor blockers
Ca ++ channel blockers
alpha blockers
End test for patients that have had a heart attack
<39 -140bpm and 7 METs >/= 40- 130bom and 5 METs
everyone else- Angina at a rate of 3
Balke-ware protocol
increase 2% grade for 1 min
then increase 1% for every min
speed 3.4 mph
benefits from training!
increased VO2
improved VT ( submax becomes easier)
improved LT
relief of secondary symp
decrease body weigh, BP, LDL, TAG, total cholesterol, inflammatory markers, blood viscosity
increase HDL
phase 1 and 2 of cardiac rehab
FITT*****